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interleukin-2 (Interking)

✓ Approved

Shenzhen Neptunus · IL2RA · 重组蛋白

什么是 interleukin-2?

interleukin-2 是一种重组蛋白,由Shenzhen Neptunus研发。该药已获批,用于治疗相关适应症,给药途径:Injectable (Others)、Intravenous (IV)、Subcutaneous Injection。

药物档案

商品名Interking
公司Shenzhen Neptunus
药物类别重组蛋白
分子靶点IL2RA
给药途径Injectable (Others), Intravenous (IV), Subcutaneous Injection
状态Approved

作用机制

分子靶点

interleukin-2 作用于 1 个分子靶点:

IL2RAinterleukin 2 receptor subunit alpha (IL2R, TCGFR)
需要更深入的分析?Noah AI 可解释复杂机制并与同类药物比较。

治疗适应症

interleukin-2 针对 15 个适应症,涉及 4 个治疗领域。

治疗领域疾病/病症分期
Neoplasms benign, malignant and unspecified (incl cysts and polyps)Adenosquamous cell lung cancer✓ Approved
Neoplasms benign, malignant and unspecified (incl cysts and polyps)Bladder cancer✓ Approved
Neoplasms benign, malignant and unspecified (incl cysts and polyps)Breast cancer✓ Approved
Infections and infestationsHepatitis B✓ Approved
Infections and infestationsLeprosy✓ Approved

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相关研究文献

PubMedCureus2026-09-10

Comparative Efficacy of Interleukin Inhibitors and JAK Inhibitors in Moderate to Severe Plaque Psoriasis.

Daily Alexandra A, Danchulis London L, Krulikowski Kiarra K, Shectman Brittany B et al.

Plaque psoriasis is a chronic autoimmune skin condition characterized by erythematous, scaly plaques that commonly affect the scalp, trunk, and extensor surfaces. As the most prevalent form of psoriasis, it results from immune system dysregulation, particularly involving cytokines. Cytokines such as interleukin-17 (IL-17) and interleukin-23 (IL-23), as well as downstream Janus kinase (JAK)/tyrosine kinase 2 (TYK2) signal pathways, lead to accelerated skin cell turnover. While an expanding array of treatment options exists, including topical agents, conventional oral medications, and targeted biologics, gaps persist in optimizing long-term management, such as improving access to medications and balancing efficacy with patient preference. Effective, practical, and acceptable long-term treatment options are needed. Injectable interleukin inhibitors, such as risankizumab (Skyrizi©) and bimekizumab (Bimzelx©), have demonstrated significant efficacy in treating moderate to severe plaque psoriasis. However, the recent FDA approval of deucravacitinib (Sotyktu©), an oral TYK2-selective JAK inhibitor, presents a promising alternative that may represent an important oral alternative for selected patients, particularly for patients who are needle-averse, have limited access to biologic infusions, or prefer oral therapies. This structured narrative review evaluates the current evidence surrounding leading IL-17 and IL-23 inhibitors and evaluates the potential role of JAK/TYK2 inhibition in addressing gaps in care. By comparing efficacy, safety profiles, and practical considerations across these agents, this review highlights the need for further longitudinal studies and real-world evidence to guide individualized treatment planning and broaden access to patient-centered care.

PMID 42719781
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PubMedStress biology2026-09-10

Lycopene mitigates T-2 toxin-induced systemic inflammation and oxidative stress in association with gut microbiota and SCFAs regulation in mice.

Adam Saber Y SY, Ennab Wael W, Zhu Cuipeng C, Yuan Long L et al.

T-2 toxin is a trichothecene mycotoxin produced by Fusarium species and can cause disease after contaminated food or feed ingestion. This study aimed to investigate the impact of lycopene (Ly) on systemic inflammation, oxidative stress, and dysbiosis in mice exposed to T-2 toxin. A total of 20 male BALB/c mice (6 weeks old, weighing 23.5 ± 0.32 g) randomly divided into four groups (n = 5): Control (Cont), Ly, T-2, and T-2 + Ly groups. Findings revealed that Ly enhanced the body weight, feedd, and water intake that have been reduced by T-2. Ly increased the villus height (VH) and VH: crypt depth (CD) and decreased CD and villus width (VW) in ileum compared to the T-2 group. In alpha diversity, Staphylococcus and Corynebacterium were the most prevalent in Cont group; Staphylococcus, and Lactobacillus, were the most prevalent in T-2 group; Candidatus-Arthromitus and Staphylococcus were the most prevalent in T-2 + Ly group. Moreover, T-2 mice had significantly (p < 0.05) greater levels of interleukin-1 beta (IL-1β), interferon gamma (IFN-γ), Interleukin-17 (IL-17), and tumor necrosis factor-alpha (TNF-α) than Cont, while the Ly significantly (p < 0.05) lowered their concentration. The T-2 mice had significantly (p < 0.05) higher levels of reactive oxygen species (ROS) and malondialdehyde (MDA), while their catalase (CAT),, glutathione (GSH), adenosine triphosphate (ATP), and superoxide dismutase 1(SOD1) activities were significantly (p < 0.05) lower than Cont. Ly significantly (p < 0.05) restored their concentration. Moreover, there was a significant reduction (p < 0.05) of hexanoic, butyric, acetic, pentanoic, and Heptanoic acids in the T-2 mice compared to Cont. In contrast, treatment with Ly was found to significantly restore their levels compared to T-2 group. We conclude that Ly administration in the context of T-2-induced toxicity may help attenuate systemic oxidative stress and inflammation, possibly through modulation of gut microbiota and fecal short-chain fatty acids (SCFAs).

PMID 42720699
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PubMedGeneral physiology and biophysics2026-09-10

Cornuside attenuates inflammatory response injury in mice with ovalbumin-induced allergic rhinitis.

Wang Hui H, Huang Linghan L

Allergic rhinitis (AR) is characterized by inflammation of the nasal mucosa caused by an allergic reaction. Cornuside has been reported to possess anti-allergic properties. This study aims to investigate whether cornuside can suppress AR. Mice were induced by ovalbumin (OVA) to establish AR models. The AR symptoms including sneezing and nasal rubbing were recorded, and the eosinophils in mouse mucosal tissues were analyzed by hematoxylin and eosin staining. Total leucocytes in nasal lavage fluid (NLF) were quantified under a hemocytometer. Wright's Giemsa staining was utilized to quantify eosinophils, lymphocytes, and neutrophils in NLF. Enzyme-linked immunosorbent assay was conducted to detect histamine, serum cytokines (OVA-specific IgE, OVAspecific IgG1, interleukin-4, and leukotriene C4), and serum inflammation cytokines (tumor necrosis factor-α, interleukin-6, interleukin-13, and interleukin-17). Cornuside administration significantly reduced sneezing and nasal rubbing behaviors in mice with AR. Mice with AR exhibited more inflammatory cells and higher levels of histamine, serum cytokines, and inflammatory cytokines in the blood or NLF than the control group. However, these upregulations were all reversed by the administration of cornuside. In addition, we found that cornuside treatment improved the inflammatory condition of mice with AR dose-dependently Cornuside can attenuate the inflammatory response in mice with AR.

PMID 42717790
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PubMedInternational journal of cosmetic science2026-09-10

Chaves thermal spring water formulation: Substantiating anti-ageing and anti-inflammatory effects.

Rocha Pedro Emanuel PE, Faustino Margarida M, Pedrosa Silvia Santos SS, Alves Maria José MJ et al.

Thermal spring waters are known for their distinctive mineral compositions, which provide health-promoting, antioxidant, immunomodulatory and skin barrier-reinforcing effects. Historically valued for therapeutic use, these waters have recently gained renewed interest as active components in cosmetic and dermatological formulations. Previous works characterized one of Portugal's most renowned thermal waters from Chaves, revealing its potential as a dermatological base ingredient due to its anti-inflammatory effects, namely the reduction of interleukin-6 in stimulated human keratinocytes. In vivo assays further demonstrated improved skin barrier function, as evidenced by decreased transepidermal water loss and enhanced hydration without disturbing the skin microbiota. Building on these results, a new formulation was developed using Chaves thermal water as both solvent and active ingredient, combined with natural extracts of mallow (Malva sylvestris) and cucumber (Cucumis sativus), and the natural chelating agent sodium phytate to achieve a high natural index. This formulation was compared with the isolated thermal water to evaluate synergistic bioactivities. A formulation containing ~80% Chaves thermal spring water, Malva sylvestris and Cucumis sativus extracts and sodium phytate was prepared and characterized. Antioxidant activity (ABTS/DPPH), ferrous ion chelation and inhibition of matrix metalloproteinase-1, tyrosinase and elastase were evaluated. Anti-inflammatory effects were assessed in particulate matter-stimulated HaCaT keratinocytes by measuring interleukin-6 and interleukin-1α (Enzyme-Linked Immunosorbent Assay (ELISA)). Antimicrobial screening against Staphylococcus aureus and Staphylococcus epidermidis was performed using growth curves. This formulation was compared with the isolated thermal water to evaluate synergistic bioactivities. The results revealed a substantial increase in antioxidant capacity, as shown by enhanced ABTS radical scavenging activity and stronger inhibitory effects on ageing-related enzymes, including matrix metalloproteinase-1, tyrosinase and elastase. Anti-inflammatory activity was confirmed through reductions in both interleukin-1α and interleukin-6 levels, the former showing notable improvement compared with the thermal water. Overall, this study demonstrates that combining Chaves thermal spring water with synergistic natural extracts yields a multifunctional cosmetic ingredient with amplified antioxidant, anti-inflammatory and anti-ageing properties, offering strong potential for advanced skin care applications.

PMID 42717854
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PubMedNature communications2026-09-10

IL-2 mutein-engrafted antibody MHS552 selectively expands functional regulatory T cells in nonclinical models and healthy participants.

Radanović Igor I, DiDonato Michael M, Meijs Anouk C AC, Schubert David A DA et al.

Low-dose interleukin-2 (IL-2) shows potential for treating autoimmune disorders by expanding regulatory T cells (Treg), but therapeutic utility is limited by short half-life and off-target effects. Here, we describe the molecular design, nonclinical development, and first-in-human study of MHS552, an IL-2 mutein-engrafted antibody designed to selectively expand Treg via high-affinity IL-2 receptors. In vitro, MHS552 selectively induces IL-2 signaling in Treg from healthy donors and autoimmune patients. In cynomolgus monkeys, MHS552 produces dose-dependent Treg expansion, with modest increases in conventional T cells (Tconv) at higher doses. In a randomized, double-blind, placebo-controlled, single-ascending-dose trial in 60 healthy participants (EudraCT 2018-004233-33), evaluating safety and tolerability as the primary objective and pharmacokinetics as the secondary objective, intravenous and subcutaneous administration of MHS552 is well tolerated at lower doses and shows a predictable pharmacokinetic profile. However, a serious adverse event (SAE) is reported at the highest subcutaneous dose. Both administration routes result in dose-dependent Treg expansion, with up to 6-fold increase in total Treg and 60-fold increase in CD25hi subset, without significant conventional T cell activation. Exploratory analyses confirm selective Treg activation, with expanded Treg retaining their suppressive function ex vivo. These findings support IL-2-based therapeutics for autoimmune disorders, while the SAE highlights the need for careful safety evaluation.

PMID 42716926
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PubMedFrontiers in medicine2026-09-10

Case Report: Balancing immunosuppression and infection-intravenous immunoglobulin in anti-HMGCR immune-mediated necrotizing myopathy complicated by severe pneumonia.

Fu Jing J, Zhao Qiong Q, Lou Jing-Bo JB, Tang Gui-Hua GH et al.

A 65-year-old male with a 3-year history of atorvastatin use presented with progressive proximal muscle weakness, dysphagia, and markedly elevated creatine kinase (2,277 U/L). Initially misdiagnosed with polymyositis, he deteriorated with severe pneumonia, septicemia, and type I respiratory failure requiring mechanical ventilation. Interleukin-6 rose from 12.83 to 610.9 pg/mL during sepsis. After confirmation of anti-HMGCR antibody positivity (18 arbitrary units [AU], cutoff >10 AU) via line blot assay, we discontinued methotrexate, maintained methylprednisolone at 40 mg, and initiated intravenous immunoglobulin (IVIG) 0.4 g/kg/day for 5 days with broad-spectrum antibiotics. Within two weeks, proximal muscle strength (MRC scale) improved from 2/5 to 4/5, IL-6 normalized to 8.56 pg/mL, and respiratory failure resolved. This case suggests that IVIG may serve as a safe immunomodulatory bridge in critically ill IMNM patients with severe infections.

PMID 42718719
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